Borna virus and bipolar

Its name derived from the town Borna in Saxony, Germany, where a large number of horses died during an epidemic in Borna disease virus exists world-wide in horses, sheep, cattle, cats, dogs, and ostriches. This transmission through nasal, salival, or conjunctival secretions is currently assumed. It spreads trans-synaptically and intra-axonally towards olfactoric structures before moving onto the limbic system.

The proteins located on the BDV are demonstrated to represent the major antigenic components in BDV infection to which the humoral immune response is directed. The matrix M protein is involved in virus attachment and aggregates to a tetrameric form by strong hydrophobic interactions. Bornavirus infection can be diagnosed from a small blood sample ml citrated blood or serum, children 1 ml using special assays, such as ELISA formats.

Patients diagnosed with BDV infection are treated with a drug that has been approved for the past 40 years for treatment of influenza A virus, which has the active ingredient of amantadine sulphate. Those with this disease may develop behavioural disorders and show signs such as being aggressive or irritable. When BDV leads to encephalitis, this may cause psychiatric symptoms in patients who recover from the initial infection.

One study investigated the use of antiviral medication in patients with both BDV and depression ; the participants had been diagnosed with either major depression or bipolar disorder. It was found that the medication successfully fought the infection, while also reducing depressive symptoms. There was a clear difference between the anti-depressant effect of the drug compared to the placebo.

This suggests that borna virus may contribute towards symptoms of depression. Borna virus patients may be treated with an antiviral drug such as amantadine. Bioresonance therapy has been found to be effective in removing the symptoms of a borna virus infection by supporting the immune system and the gut, as well as helping the body to detoxify. Borna disease virus not only affects animals, but can also cause severe neurological symptoms in humans, which may even be fatal.

Even if a serious reaction is not experienced, the presence of this virus may contribute towards mental health problems such as depression. The interpretation is that the immune cells must be causing the inflammation that is causing the schizophrenia. If they had the diagnostic tools to see what it actually going on they would find that the immune system is targeting infections in the cells. Removing the infection would eliminate the problem. The Borna virus was first discovered in horses in Germany and the Germans have done many of the best studies on this major public health problem.

They have tested for the virus in depressed patients and found it, treated the virus and determined it was eliminated in lab tests. They then showed the depression was significantly reduced in most of the patients. There are hundreds of studies on the Borna virus posted on www. Those afflicted should carry the book in this posting into the psychiatrists office so they can educate them. It took a hundred years from publication of a monogram at Johns Hopkins suggesting that ulcers were caused by H.

Pylori before psychiatrists stopped sending people to therapy for them. And only an article in the National Enquirer caused enough uproar to wake up the scientific community. Not only did the researchers find no relationship between mental illness and bornavirus, they found no evidence of active or historical infection with BDV in any of the subjects.

In a commentary in the same issue of the journal, Michael B. Oldstone, MD, an expert in molecular virology and central nervous system infections at the Scripps Research Institute, observes that the design and experimental procedures carried out in the Hornig study provide a gold standard for investigating links between persistent viral infection and human disease.

CII director, W.